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Nonlinear dynamics and stochastic fluctuations in gene regulatory networks

Alexander Karlshofer, Jan Ribbe

Individual cells within an isogenic cell population can launch different genetic programs that determine cell fate. Phenotypic bistability in genetically identical populations controls development of eukaryotic cells, virus latency, and bacterial sporulation and competence for DNA transformation. We are studying the development of competence for DNA transformation in Bacillus subtilis.

Genereg Small

When cell density is high a well-defined fraction of cells expresses proteins that are required to assemble the DNA import machine; this competent fraction of the bacterial cell population expresses the master regulator for DNA uptake proteins, ComK, at high level. A genetic switch is implemented by nonlinear amplification of the master regulator ComK: in the B-state low expression of ComK is low and in the K-state expression of ComK is high. Using a fluorescent reporter for the master regulator we have shown that expression in the B-state is noisy and that that basal expression sets a switching window. We are currently combining quantitative microscopy with nonlinear dynamics simulations to investigate how perturbations of the regulatory network and external conditions influence stability and switching kinetics.

Techniques

  • Quantitative image analysis 

Collaborations

Erwin Frey, LMU München

Joachim Rädler, LMU München

Publications

  • Maier, B. Competence and transformation. in Bacillus: Cellular and Molecular Biology, ed. Peter Graumann, Caister University Press (2011)

  • Maier, B. Stochastische Differenzierung als bakterielle Überlebensstrategie. Biospektrum 2/2010